You Searched For: Clinical+Diagnostic+Supplies

Catalog Number: (V-027-1ML)

Supplier: Merck

Description: A certified reference solution suitable for use as an internal standard for quantitation of O-desmethylvenlafaxine levels in urine or isotope dilution methods by LC/MS or GC/MS for clinical toxicology, urine drug testing, or forensic analysis.

UOM: 1 * 1 mL

Supplier: RATIOLAB

Description: All-purpose PE bags with self-adhesive strip for clinical and laboratory use.

Supplier: Merck

Description: For staining of blood and bone marrow smears and clinical-cytological specimens

MSDS

Catalog Number: (LIOF26490)

Supplier: LIOFILCHEM

Description: VTM is suitable for collection and preservation of clinical specimens containing viruses, including SARS-CoV-2 (COVID-19), Chlamydia spp., Mycoplasma spp. and Ureaplasma spp.

UOM: 1 * 100 items

Supplier: HUBEI ORIENT INTL TRADING

Description: Disposable bouffant caps made from non woven PP. Ideal for use in non hazardous environments.

Supplier: HUBEI ORIENT INTL TRADING

Description: These disposable shoe covers are made of soft and durable CPE.

Catalog Number: (LIOF96147)

Supplier: LIOFILCHEM

Description: Identification of the main pathogenic microorganisms directly from clinical specimens.

UOM: 1 * 1 items

Catalog Number: (BOSSBS-10044R-A750)

Supplier: Bioss

Description: NDV is a contagious and fatal viral disease affecting most species of birds. Clinical signs are extremely variable depending on the strain of virus, species and age of bird, concurrent disease, and preexisting immunity. Four broad clinical syndromes are recognised by scientists. They are Viscerotropic velogenic, Neurotropic velogenic, Mesogenic, and Lentogenic. NDV is so virulent that many birds die without showing any clinical signs. A death rate of almost 100 percent can occur in unvaccinated poultry flocks. NDV can infect and cause death even in vaccinated poultry. Fortunately NDV has not infected domestic chicken flocks in the United States since the last outbreak was eradicated in 1974.

UOM: 1 * 100 µl

Catalog Number: (BOSSBS-4807R-HRP)

Supplier: Bioss

Description: Defects in ATXN3 are the cause of spinocerebellar ataxia type 3 (SCA3) ; also known as Machado-Joseph disease (MJD). Spinocerebellar ataxia is a clinically and genetically heterogeneous group of cerebellar disorders. Patients show progressive incoordination of gait and often poor coordination of hands, speech and eye movements, due to degeneration of the cerebellum with variable involvement of the brainstem and spinal cord. SCA3 belongs to the autosomal dominant cerebellar ataxias type I (ADCA I) which are characterized by cerebellar ataxia in combination with additional clinical features like optic atrophy, ophthalmoplegia, bulbar and extrapyramidal signs, peripheral neuropathy and dementia. The molecular defect in SCA3 is the a CAG repeat expansion in ATXN3 coding region. Longer expansions result in earlier onset and more severe clinical manifestations of the disease.

UOM: 1 * 100 µl

Catalog Number: (BOSSBS-4807R-A750)

Supplier: Bioss

Description: Defects in ATXN3 are the cause of spinocerebellar ataxia type 3 (SCA3) ; also known as Machado-Joseph disease (MJD). Spinocerebellar ataxia is a clinically and genetically heterogeneous group of cerebellar disorders. Patients show progressive incoordination of gait and often poor coordination of hands, speech and eye movements, due to degeneration of the cerebellum with variable involvement of the brainstem and spinal cord. SCA3 belongs to the autosomal dominant cerebellar ataxias type I (ADCA I) which are characterised by cerebellar ataxia in combination with additional clinical features like optic atrophy, ophthalmoplegia, bulbar and extrapyramidal signs, peripheral neuropathy and dementia. The molecular defect in SCA3 is the a CAG repeat expansion in ATXN3 coding region. Longer expansions result in earlier onset and more severe clinical manifestations of the disease.

UOM: 1 * 100 µl

Catalog Number: (ICNA091003122)

Supplier: MP Biomedicals

Description: Basal Medium Eagle is suited for the isolation and cultivation of non-fastidious and fastidious microorganisms from a variety of clinical and non-clinical specimens. It can also be used as a selective isolation medium by adding antimicrobial agents.

UOM: 1 * 10 L

Catalog Number: (BOSSBS-4807R-CY5)

Supplier: Bioss

Description: Defects in ATXN3 are the cause of spinocerebellar ataxia type 3 (SCA3) ; also known as Machado-Joseph disease (MJD). Spinocerebellar ataxia is a clinically and genetically heterogeneous group of cerebellar disorders. Patients show progressive incoordination of gait and often poor coordination of hands, speech and eye movements, due to degeneration of the cerebellum with variable involvement of the brainstem and spinal cord. SCA3 belongs to the autosomal dominant cerebellar ataxias type I (ADCA I) which are characterized by cerebellar ataxia in combination with additional clinical features like optic atrophy, ophthalmoplegia, bulbar and extrapyramidal signs, peripheral neuropathy and dementia. The molecular defect in SCA3 is the a CAG repeat expansion in ATXN3 coding region. Longer expansions result in earlier onset and more severe clinical manifestations of the disease.

UOM: 1 * 100 µl

Catalog Number: (BOSSBS-4807R-CY5.5)

Supplier: Bioss

Description: Defects in ATXN3 are the cause of spinocerebellar ataxia type 3 (SCA3) ; also known as Machado-Joseph disease (MJD). Spinocerebellar ataxia is a clinically and genetically heterogeneous group of cerebellar disorders. Patients show progressive incoordination of gait and often poor coordination of hands, speech and eye movements, due to degeneration of the cerebellum with variable involvement of the brainstem and spinal cord. SCA3 belongs to the autosomal dominant cerebellar ataxias type I (ADCA I) which are characterized by cerebellar ataxia in combination with additional clinical features like optic atrophy, ophthalmoplegia, bulbar and extrapyramidal signs, peripheral neuropathy and dementia. The molecular defect in SCA3 is the a CAG repeat expansion in ATXN3 coding region. Longer expansions result in earlier onset and more severe clinical manifestations of the disease.

UOM: 1 * 100 µl

Supplier: Merck

Description: The Giemsa’s Azure Eosin Methylene Blue solution can be used for the staining of blood and bone marrow smears, paraffin sections and clinical-cytological specimens.

MSDS

Catalog Number: (BOSSBS-4807R-A350)

Supplier: Bioss

Description: Defects in ATXN3 are the cause of spinocerebellar ataxia type 3 (SCA3) ; also known as Machado-Joseph disease (MJD). Spinocerebellar ataxia is a clinically and genetically heterogeneous group of cerebellar disorders. Patients show progressive incoordination of gait and often poor coordination of hands, speech and eye movements, due to degeneration of the cerebellum with variable involvement of the brainstem and spinal cord. SCA3 belongs to the autosomal dominant cerebellar ataxias type I (ADCA I) which are characterized by cerebellar ataxia in combination with additional clinical features like optic atrophy, ophthalmoplegia, bulbar and extrapyramidal signs, peripheral neuropathy and dementia. The molecular defect in SCA3 is the a CAG repeat expansion in ATXN3 coding region. Longer expansions result in earlier onset and more severe clinical manifestations of the disease.

UOM: 1 * 100 µl

Catalog Number: (BOSSBS-4807R-A555)

Supplier: Bioss

Description: Defects in ATXN3 are the cause of spinocerebellar ataxia type 3 (SCA3) ; also known as Machado-Joseph disease (MJD). Spinocerebellar ataxia is a clinically and genetically heterogeneous group of cerebellar disorders. Patients show progressive incoordination of gait and often poor coordination of hands, speech and eye movements, due to degeneration of the cerebellum with variable involvement of the brainstem and spinal cord. SCA3 belongs to the autosomal dominant cerebellar ataxias type I (ADCA I) which are characterized by cerebellar ataxia in combination with additional clinical features like optic atrophy, ophthalmoplegia, bulbar and extrapyramidal signs, peripheral neuropathy and dementia. The molecular defect in SCA3 is the a CAG repeat expansion in ATXN3 coding region. Longer expansions result in earlier onset and more severe clinical manifestations of the disease.

UOM: 1 * 100 µl